Pulmonary Embolism Support Group
By far the most common form of pulmonary embolism is a thromboembolism, which occurs when a blood clot, generally a venous thrombus, becomes dislodged from its site of formation and embolizes to the arterial blood supply of one of the lungs. Symptoms may include difficulty breathing, pain during breathing, and more rarely circulatory instability and death.
AprilPEShower
With the understanding that every case is different, doctors want patients stable at very different levels, that stable is an ambiguous word, etc., . . . for those who achieved stability (stable INR) what dossage of warfarin/coumadin were/are you taking?
Comparison sometimes helps to show that you are not that different than others. If you do not believe there is any value to this, then shoot it down, the information is what can help others.
Despite my INR still bouncing around wildly as I approach the 3 month mark, it looks like 8 mg of coumadin a day is where I should be.
Comparison sometimes helps to show that you are not that different than others. If you do not believe there is any value to this, then shoot it down, the information is what can help others.
Despite my INR still bouncing around wildly as I approach the 3 month mark, it looks like 8 mg of coumadin a day is where I should be.
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I need 14 mgs a day at this point. I had been at 15 mgs for a while, a couple of years ago, and 11-12 mgs for a while but over time things change so the dosage gets adjusted. This has been going on for over 6 years with me. So honestly, I don't know that there's ever one dosage that works, particularly if you're on the stuff for the long term.
From the matter of perspective, I used to know a lady whose husband took 2mg a WEEK. She was floored when she found out how much I took. From her POV, my dose was completely astronomical.
Everyone's different.
Anyone else have a different color? Do they make a 10? I have to take 2 fives tonight.
1mg are pink, BTW.
Any other colors we know of?
Gotta love Google: http://www.coumadin.com/html/atpharmacy.htm
Mike
Jewish bleeding disease/disorder is an old name for Factor XI deficiency, which is now called Hemophilia C. It's an autosomal dominant gene which passes to both male and female offspring. Homozygotes have much more severe symptoms than heterozygotes. For those of you who didn't major in genetics, that means the gene is on a regular chromosome, unlike Hemophilias A & B, which are on the X-chromosome. Both men and women have two copies of this gene, and either gender can have one good and one bad copy of the gene or two bad copies. Those with two bad copies usually bleed more than those with one. It was first discovered and is still most prevalent in Ashkenazi Jewish populations, hence the early-1900's moniker.