Pulmonary Embolism Support Group
By far the most common form of pulmonary embolism is a thromboembolism, which occurs when a blood clot, generally a venous thrombus, becomes dislodged from its site of formation and embolizes to the arterial blood supply of one of the lungs. Symptoms may include difficulty breathing, pain during breathing, and more rarely circulatory instability and death.
infection."
http://www.copacamu.org/IMG/pdf/InfectionetVT.pdf
There are other studies connecting acute infection (UTI) as a possible precursor to a person having a DVT or PE.
The main contributors, of course, remain: genetic, sitting too long (for some people), surgery, birth control pills etc.
The only reason UTIs as a precursor are obscure is simply the lack of studies done. That's all.
And of course, it doesn't mean that everyone who gets an UTI will get a DVT!! That goes without saying. But it happens.
Furthermore, I had always had doubts about the UTI, since there were very bacterias identified and because I really had no other symptom than the burning sensation and that went away the next day, but the right flank pains and general "I'm not feeling good" feelings remained. And because this was the only "UTI" I ever had. Since the PE, I've been thinking that it may just have been the PEs with the typical vague symptoms.
It's just that if the UTI was the cause, it may not have been an idiopathic PE and may mean I can eventually come off the coumadins.
"We do not know whether the transient increase in risk is due to a short-term alteration of endothelial function or to other mechanisms such as white-cell activation or dehydration," they added. "However, infection is always associated with inflammation, and C-reactive protein is raised in patients diagnosed in a primary-care setting with respiratory tract infection."
Other researchers have identified increased risk for venous thromboembolism in patients with non-infectious inflammatory conditions such as inflammatory bowel disease and rheumatoid arthritis, the authors noted."
http://www.medpagetoday.com/HematologyOncology/Hematology/2965
However, the authors were careful to add (and so would I)--
"confounding factors could not be completely ruled out."
Just because it's possible doesn't mean it will happen with everyone.
And just because that causal relationship is obscure that's something that is always a problem.
What isn't looked for---- isn't found.
If I'm interpreting that right---- any UTIs I ever had, as bad as they were, never forced/allowed me to stay home from work or disengage from 'normal' activities, esp. after treatment was started. The first days might have been 'iffy' when that horrible urge to pee was there.
Perhaps others had a different/worse strain?
"The most common nosocomial infections are of the urinary tract, surgical site and various pneumonias."
Nosocomial = hospital acquired
I am on low dose amoxicillin in order to prevent the UTIs that I was getting over and over, despite tx. I sent my doctor the journal article that advised that, for certain populations and he complied.
Males, due to a different anatomy, have far fewer UTIs than females so we'll hope that his have gone for good since it was probably nosocomial.
The much touted antibiotic resistance craze that's sweeping the medical community now results in insufficient tx in many times, in my opinion. Rant follows: get rid of food animal abx and maybe 'we' don't have to worry so much. Rant over.