Atrial Fibrillation (AFib) Support Group
Atrial fibrillation (AF or afib) is an abnormal heart rhythm (cardiac arrhythmia) which involves the two small, upper heart chambers (the atria). Heart beats in a normal heart begin after electricity generated in the atria by the sinoatrial node spread through the heart and cause contraction of the heart muscle and pumping of blood.
Mine put me on 50 mg. 2x day. My rate dropped to low 40's right away. So he said, 25mg. 2x day. Rate was in upper 40's....and I was so sleepy.
Then he put me on extended release 50mg. to take at night...rate still too slow, felt awful.....and I was still going into afib. But, I sure did sleep well.
I was certain the Metoprolol was not necessary for me to take all the time. I asked to be taken off. Dr. said, No, I needed a pace maker so I could tolerate the Metoprolol. He said I had Bradycardia....a permanent low heart rate. This seemed crazy to me.
I went for a 2nd opinion. This Dr. told me to take the Metoprolol only when I went into afib. I am symptomatic and know when I go into afib...and my rate goes to 140. This has worked for me for 4 yrs. I also have Rythmol...an antiarrhythmic, that I can take with the Metoprolol...if I want to.
Even when I take 25 mg. at the onset of afib, I am still a bit groggy for the day. Each episode is different in how and when I convert.
I also take Pradaxa~
Dr. says, keep exercising.......ya, right, I have to plan ahead for the strength to get up from the chair I'm sitting in most of the time.
Might want to check that out. Im taking thyroxine now and feel alot better....theres some older post on here about afib and hypothyroidism.
Hope you feel better
Vinny
After my first ablation I was put on the same schedule as you (50mg 2X day). I would take it at night, sleep for 12+ hours. Get up and have breakfast (up for about an hour) and take the next dose, then nap until noon. Get up and eat again, then nap from about 1 to 6. Get up eat again, in bed by ~7.
Repeated this for about two weeks. Decided I could not function on this dosage/meds. So at my checkup, DR scaled back to 25mg 2X a day. It still makes me fatigue (after 4 years), and I'm down to 12.5 mg 1x a day.
Robert
Ask about switching to bystolic.
I, too, was put on metoprolol 50mg per day after being cardioverted after my very first of the 3 afib episodes I had (came on at night while sleeping). 3 months later, the doctor took me off of it, wanting to see if I could go without it. I lasted 2 months and then I fell back into afib - in the middle of the night while sleeping. One 50mg metoprolol tablet (per doctor's instructions terminated it.
I then tried (with the doctor's concurrence) 25mg per day and lasted 2 months with that before falling back into afib (again at night while sleeping soundly).
In the ER with that 3rd episode of afib, they gave me 300mg of Flecainide which terminated the episode in 2 hours. Since then, the doctor has had me on Flecainide 50mg x 2 per day and I have not had any more episodes for over 13 months hence.
Flecainide has exerted very little in side effects - doesn't make me feel tired. It does tend to slow my heart rate and keeps my peak exercise heart rate lower than it would be otherwise. My doctor says Flecainide is the best antiarrhythmic on the market. But he did give me an echo and a nuclear stress/exercise study as they prefer to do with all Flecainide patients as the heart structure has to be sound for it to be safe.
Beta blockers don't cause sudden death, in fact they prevent it... from National Institutes of Health:
"Beta blockers have been shown to reduce the risk of sudden cardiac death in more than 50 randomized trials involving more than 55,000 patients. Relative reductions (vs. placebo) in cardiac death in some of these trials ranged from 30 to 50%. These reductions are substantially greater than trials of other drug classes including angiotensin-converting enzyme inhibitors. However, not all beta blockers confer equal benefit to patients at risk of sudden cardiac death."
From a recent PubMed/NIH URL:
"CONCLUSION:
"Beta-blockers reduce the risk of sudden cardiac death (SCD) by 31%, cardiovascular death (CVD) by 29% and all-cause mortality by 33%. These results confirm the mortality benefits of these drugs and they should be recommended to all patients similar to those included in the trials."
I think you are talking about Multaq:
"As we know, now, since being approved by the FDA in 2009 and soon becoming an often-prescribed atrial fibrillation drug, Multaq has been associated with increased risks of serious side effects, including liver failure, interstitial lung disease (ILD), stroke, and sudden cardiac death."
"In the February 2014 edition of JAMA Internal Medicine one finds this rather hard-hitting article, "Dronedarone for Atrial Fibrillation: The Limited Reliability of Clinical Practice Guidelines".
http://www.drug-injury.com/druginjurycom/2014/03/multaq-increased-risks-lung-damage-liver-failure-heart-attack-cardiac-death-stroke-side-effects.html
petey