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Stroke characteristics and http://www.selleckchem.com/products/poziotinib-hm781-36b.html outcomes recorded were stroke arterial territory location, baseline and 24-h NIHSS score,[10] Oxfordshire Community Stroke Project classification,[13] symptomatic ICH, duration of admission and discharge mRS score. We determined incidence of ICH from follow-up CT scan and defined it as symptomatic if the patient had any associated neurological deterioration attributable to the presence of parenchymal haemorrhage by the treating neurologist.[14] Neurological improvement was defined as an improvement of 4 points on NIHSS score, or a score of 0 or 1 at 24?h.[7, 11] NIHSS and mRS assessments were performed by fully accredited neurologists or neurology trainees. Statistical analyses were performed on SPSS program (SPSS, Chicago, IL, USA). Data was analysed using t-test or Mann�CWhitney test as deemed appropriate. The impact of code stroke on clinical outcomes was tested through multiple logistic regression adjusting for baseline variables. A P-value of http://www.selleckchem.com/products/ganetespib-sta-9090.html 56% of patients were males, and the median NIHSS score was 13.[8-19] The baseline characteristics of patients in the code stroke era and pre-code stroke era were well matched (Table?1). The only observed difference between the two groups was a prior history of stroke (P = 0.01). The median pre-code stroke group onset-to door time was 61.50 (interquartile range (IQR) 49.0�C73.8) min, and the median code stroke onset-to-door time was 72.0 (IQR 56.0�C111.50) min (P http://www.selleck.cn/products/cobimetinib-gdc-0973-rg7420.html stroke vs 72.0 (IQR 50.5�C93.5) min code stroke, P