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A second patient had a hepatic artery, which could not be visualized with postoperative ultra-sound due to body habitus. He underwent an angiogram revealing normal arterial flow, but developed a large groin hematoma, which was managed conservatively. By 8 months from transplant this patient had developed excessive weight loss (weight = 60.2 kg, BMI = 20) and required dietary counseling. The patient reported he felt well but simply had no interest in eating as he never felt hungry. This patient subsequently developed a late hepatic artery thrombosis at approximately 11 months posttransplant. This resulted in multiple hepatic abscesses requiring drainage and prolonged http://www.selleckchem.com/products/SB-431542.html antibiotics. These have resolved and the patient currently has normal allograft function, and a stable BMI of 23. A third complication involved a patient with one episode of steroid-resistant rejection, which required treatment with thymoglobulin. There were no other episodes of steroid resistant rejection, and tacrolimus dosing did not require additional http://www.selleck.cn/products/ipi-145-ink1197.html monitoring or adjustment, compared to standard patients. Although outcomes following LT for selected patients with obesity are broadly similar, posttransplant obesity and complications of obesity are common. The ideal approach to the management of the obese liver transplant patient remains unknown. Our current study has several important findings pertinent to management of obesity in liver transplant recipients. The first notable result of this study is that an intensive noninvasive pretransplant weight loss protocol was effective for the majority of our patients in achieving http://www.selleckchem.com/products/byl719.html pretransplant weight loss to a BMI of