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Infected house finches held at both temperatures showed higher levels of IL-6 activity than uninfected controls (Fig.?3; infection status: F1,22?=?40��91, P? http://www.selleck.cn/products/azd4547.html housed at subthermoneutral temperatures (Fig.?3; infection status*treatment: F1,22?=?8��65, P?=?0��008). Infection status significantly influenced body temperature (Fig.?4; F1,26��1?=?9��56; P?=?0��005), with infected birds from both thermal treatments increasing their body temperature post-inoculation relative to uninfected birds. The subthermoneutral treatment tended to result in lower average body temperatures regardless of infection status (F1,26��1?=?3��44; P?=?0��08). There was no interaction between treatment and infection status on body temperature (P?>?0��92); though qualitatively, infected birds in the two treatments appear to behave distinctly. Birds in the thermoneutral treatment experienced a 2?��C increase in body temperature at day 1 post-inoculation (Fig.?4), and body temperatures remained high (on average >1?��C) for most days of infection. In contrast, infected individuals in the subthermoneutral treatment showed a moderate increase in body temperature http://www.selleckchem.com/products/AZD6244.html (average of 0��8?��C on day 1) that rapidly declined over the first few days of infection. Birds in both temperature treatments maintained http://www.selleckchem.com/products/MK-1775.html their body mass over time (treatment as main effect or in interaction with infection or time: all F??0��59). Although body mass generally decreased in infected birds during the initial stages of infection (days PI*infection status: F1,143?=?4��75, P?=?0��03), this effect did not vary with temperature treatment. Pectoral muscle condition also decreased in infected birds over time (days PI*infection status: F1,143?=?13��3, P?