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There are some differences in nomenclature between human and veterinary dermatology. For the most part, this is likely attributable to increased knowledge in the human medical field, and therefore more detailed description of reactions seen in human medicine. In this section, we describe different cutaneous reaction patterns that are seen as a result of adverse drug reactions, but it is important to remember that cutaneous drug hypersensitivity reactions can look like any known dermatology lesions and affect any part of the body (Table?2). Specific clinical signs cannot be attributed to specific drugs because each drug can induce different clinical signs, both qualitatively http://www.selleckchem.com/products/ch5424802.html and quantitatively, depending on the individual patient (eg, Fig?2B and C). Angioedema/urticaria histopathologically is characterized http://www.selleck.cn/products/Everolimus(RAD001).html by edema. Urticarial lesions will have edema of the dermis, whereas in angioedema there is severe edema that extends from the dermis into the subcutis. Toxic epidermal necrolysis is characterized by full-thickness coagulative necrosis with minimal dermal inflammation. Seen both in animals and humans, these are edematous cutaneous reactions that result from mast cell or basophil degranulation[2] because of Type I hypersensitivity reactions. The characteristic lesion is a hive, or wheal. In the case of urticaria, the edema is confined to the dermis, whereas in angioedema the edema spreads to http://www.selleckchem.com/products/azd9291.html the subcutaneous tissue, which makes lesions less distinct. Wheals are most commonly seen on the glabrous skin, often the ventral abdomen, and lesions often persist for