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4%] and non-identical [21.3%]) (p? of g2�C3 following ABO-incompatible transplant was 28.6%, which was significantly higher than that of other transplant groups (identical [6.5%] and non-identical [6.1%]) (p? http://www.selleckchem.com/products/pci-32765.html margination or thromboses (Type II) and arterial-v3 (Type III) (10). All four cases (100%) with g3 of glomerulitis score and one case (25%) with g2 following ABO-incompatible transplants were diagnosed AMR. One case (100%) with glomerulitis of g3 following ABO-identical transplants and one of two cases (50%) with g2 following non-identical transplants were diagnosed AMR. As to i score, ptc score and v score, there was no significant difference between three transplant groups. Moreover, in 24 cases (85.7%) of ABO-incompatible transplant, C4d deposition on PTC involving no AMR was seen. Among them, five cases (17.9%) were recognized as AMR. In nine cases of ABO-identical and non-identical transplants (6/93; 6.5%, 3/33; 9.1%), C4d deposition on PTC was seen. Among them, two cases (1/93; 1.1%, 1/33; 3.0%) were recognized as AMR. Every case recognized as AMR was ameliorated by some treatments such as plasmapheresis and steroid pulse. Table?3 shows acute rejection ratio following ABO-incompatible, non-identical and identical transplantation. There was no significant difference in the ratio of normal finding involving no rejection between three transplant groups. As to AMR, there was significant difference between three transplant groups (p?