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This analysis showed that UA predicted an increased risk of cardiac and all-cause mortality across all subgroups. An interaction was observed between UA and sex �C demonstrating a stronger association with mortality in women than in men (P for interaction?=?0��059 for cardiac mortality and P for interaction?=?0��019 for all-cause mortality) �C and between http://www.selleck.cn/products/MLN8237.html UA and arterial hypertension �C demonstrating a stronger association between UA and mortality in patients without arterial hypertension than in patients with arterial hypertension (P for interaction?=?0��050 for cardiac mortality and P for interaction?=?0��064 for all-cause mortality). The results of subgroup analyses are shown in Figs?4 and 5. In this study, we investigated the pattern of association http://www.selleckchem.com/products/XL184.html between UA and mortality (cardiac and all-cause) in a large series of patients with CAD and the strength of this association in various subgroups of patients. The main findings of the study can be summarized as follows: (i) UA predicted an increased risk of cardiac and all-cause mortality independent of traditional cardiovascular risk factors, left ventricular function, CRP and renal function across the whole spectrum of patients with CAD. (ii) The association between UA and cardiac or all-cause mortality followed a ��J-shaped�� pattern with a significant increase in the risk of death in patients with the lowest and highest UA levels. UA levels between 5��17 and http://www.selleckchem.com/products/CP-690550.html a significant increase in the risk of death for UA levels either