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88, p = 0.001) were associated with increased risk of late pancreas graft failure. Contrary to its negative impact on early pancreas graft survival, transplant center volume did not affect late pancreas graft outcome. Immunological complication, acute and chronic rejection, was the leading cause of late graft failure, accounting for 63.6% of kidney and 53.4% of pancreas graft loss, respectively. In particular, compared to non-AA SPK transplant patients, AA SPK transplant patients had a higher rate of acute and chronic rejection as the reported cause of kidney (80.3% vs. 58.9%, p http://www.selleckchem.com/products/XL184.html 2.95, p = 0.008 for pancreas loss). The recipient race has no effect on patient survival (HR 1.05, 95% CI 0.80, 1.39, p = 0.72). Older recipient age (HR 1.02, 95% CI 1.01, 1.03, p = 0.001), longer duration of dialysis (HR 1.07, 95% CI 1.03, 1.11, p http://www.selleckchem.com/products/CP-690550.html long-term pancreas and kidney http://www.selleck.cn/products/MLN8237.html graft survival (HR 1.01, 95% CI 0.82, 1.26, p = 0.90 for pancreas, and HR1.17, 95% CI 0.94, 1.45, p = 0.16 for kidney, respectively), but the bladder drainage was associated with increased risk for death (overall death and death with graft function) after adjusting for the effects of transplant era (HR 1.42, 95% 1.15, 1.75, p