Liver Support and Health Community Group
This group is dedicated to medical studies and literature about the prevention of liver failures. Our goal is to save currently affected patients with liver problems. I hope new medical data will be posted to back up new science and theories.
I was always skeptical of these new testing methods and now I have been proven correct.
In recent studies, bile excretion has been a closely contested topic, due to the use of Gd-EOB-DTPA. It assesses the accumulation patterns but doesn't clearly show the hypatoxicity of the actualy acid. It's not clear whether it's an acidic or a basic response to the actual diethylenetriaminepentaacetic acid, but there is a frequency of T1 hyperintense in the bladder. This points to toxicity of actual therapy involved.
http://www.ncbi.nlm.nih.gov/pubmed/25580112
In this study:
MR cholangiography obtained with Gd-EOB-DTPA showed that there may be an association between the biliary accumulation pattern in the gallbladder and the pathological condition
In recent studies, bile excretion has been a closely contested topic, due to the use of Gd-EOB-DTPA. It assesses the accumulation patterns but doesn't clearly show the hypatoxicity of the actualy acid. It's not clear whether it's an acidic or a basic response to the actual diethylenetriaminepentaacetic acid, but there is a frequency of T1 hyperintense in the bladder. This points to toxicity of actual therapy involved.
http://www.ncbi.nlm.nih.gov/pubmed/25580112
In this study:
MR cholangiography obtained with Gd-EOB-DTPA showed that there may be an association between the biliary accumulation pattern in the gallbladder and the pathological condition
How do you get HAAA?
It's possible to get it from drinking too much alcohol.
It's possible to get it from picking up Hepatitis B/C
It's possible to get it from using drugs
It's possible to get it from usage of methandrostenolone
It's possible to get it from a fatty diet
As you can see many factors, including diet plays into possible HAAA problems. It's up to you how you deal with the actual disease but it's something you need to treat asap:
A study on HAAA:
Clinically, AH can be mild to fulminant and transient to persistent and precedes the onset SAA. It is assumed that immunologic dysregulation following AH leads to the development of SAA. Several observations have been made to elucidate the immune mediated injury mechanisms, ensuing from liver injury and progressing to trigger bone marrow failure with the involvement of activated lymphocytes and severe T-cell imbalance. HAAA has a very poor outcome and often requires bone marrow transplant (BMT). The findings of immune related myeloid injury implied the use of immunosuppressive therapy (IST) and led to improved survival from HAAA.
References:
http://www.ncbi.nlm.nih.gov/pubmed/25587471
http://www.ncbi.nlm.nih.gov/pubmed/25578652
http://www.evolutionary.org/dianabol-methandrostenolone
http://www.ncbi.nlm.nih.gov/pubmed/25314890