I've been wondering about the same thing.. let me know what you find out xxx

One of the recent controversies with substantial clinical interest is the role of HPV in pathogenesis of ovarian cancer. The available highly conflicting data are based on analysis of 175 ovarian carcinomas so far. As an attempt to further elucidate this issue, the first systematic study of HPV detection in ovarian cancer was carried out using a highly sensitive general primer PCR (confirmed by hybridization for low- and high-risk HPV types separately) in a series of 98 histologically and clinically well-characterized epithelial ovarian malignancies. Despite the high (fg) sensitivity and a wide HPV type coverage of the technique used, all 98 ovarian carcinomas failed to demonstrate any signs of HPV DNA whatsoever. The preexisting 12 reports comprising a total of 175 ovarian tumors analyzed for HPV were summarized, giving highly discrepant results (i.e., detection rates from 0 to 100%) with the overall HPV DNA detection rate of 25.7%. The reasons for these discrepant findings are most probably technical. Our data are consistent with those of the majority of the most recent reports failing to disclose HPV DNA in ovarian neoplasia. The present completely negative results make the authors inclined to conclude that HPV is highly unlikely to play any causal role in the pathogenesis of epithelial ovarian neoplasia.

Thanks Anita.. but none of this sounds conclusive or convincing to me yet.. a bit technical perhaps for my chemo brain at the mo. I have papiloma type moles on my neck and they change all the time along with the chemo.. getting smaller and larger.. more and less..etc.. The doctors say that is to be expected.. but keep forgetting to ask them why.. Must do so next time.. Will read your reply again in the morning after a good nights sleep.. Is HPV the papiloma virus or am I barking at the wrong tree here.... sweetest dreams..

halli I was told 2 weeks ago that the HPV virus has links to Ovarian cancer, I do appriciate aniitas story but that does notsit with me either what she said. Plus my family history of breast cancer too.

I could not find anywhere on the internet that said that that was conclusive.

anita sis, internets can incluse faluse information, I get my info of doctors first hand, I do not need the internet to reserch stuff I have my doctor and specialist but thanks anyway for your imput. I hope you are coping well. and things are going well for you and your health. I have to start excerising when I can get of my bum lol. I need too.

Boarding a flight.. quick ref:

Molecular and Cellular Pathology
British Journal of Cancer (2003) 89, 672675. doi:10.1038/sj.bjc.6601172 www.bjcancer.com
Published online 12 August 2003

Detection of human papillomavirus-16 in ovarian malignancy
The study was performed at Laboratory of Genetics, Beijing Institute for Cancer Research, School of Oncology, Peking University, Beijing, China

Q-J Wu1, M Guo2, Z-M Lu2, T Li2, H-Z Qiao1 and Y Ke2

1Inner Mongolia Medical College, Hohehot Municipality 010000, China
2Laboratory of Genetics, Beijing Institute for Cancer Research, School of Oncology, Peking University, No.1 Da-Hong-Luo-Chang St. West District, Beijing 100034, China
Correspondence: Dr Y Ke, E-mail: keyang@mx.cei.gov.cn

Received 31 January 2003; Revised 28 April 2003; Accepted 5 June 2003.

Top of pageAbstract
Human papillomavirus is the causal factor for cervical cancer. However, the role of HPV infection in ovarian cancer is unclear. This study aimed to determine the presence of human papillomavirus-16 (HPV-16) in ovarian cancer tissues. Archived human ovarian cancer tissues (N=54 cases, 50 are epithelial cancer, four are nonepithelial cancer) embedded in paraffin blocks were used. Controls are 30 nonmalignant ovarian tissue blocks. In situ hybridisation (ISH) and immunohistochemistry (IHC) were used to detect the presence of HPV-16 and p53 expression. In all, 52 or 36% of the epithelial ovarian tumours detected by ISH or IHC, respectively, were HPV-16 E6 positive. In contrast, only 6.7% of normal ovarian tissues were HPV-16 positive proved by ISH. Human papillomavirus-16 infection was significantly higher in cancer tissues compared to controls with an odds ratio of 16.7 (95% confidence interval [CI]=3.271.4, P0.05). p53 gene expression was detected in 42% epithelial ovarian cancers. No correlation between p53 expression and HPV-16 infection was found. The results showed the presence of HPV-16 E6 in ovarian carcinoma, suggesting that HPV infection might play a role in ovarian carcinogenesis.

Hi there..
Know I am a little late to join in on this. But I do know the HPV does not go away. Once you have it that is it, you have it. There are many many different kinds of them, and only a few of them are what they consider a high risk HPV those are the ones they make the shot for. However it's sad you can't get that shot after you are 25..darn it!
Those high risk ones they are trying to stop are the ones that can down the road develop cancer, and as we all know its not certain if you will or wont develop cancer, but this HPV can help contribute to forming cancer. Don't know if that helped. But a way to find out more is look up or ask your DR. about info on the high risk HPV types and what they can do down the road to your body.
Lots of hugs
Renee